Studiebog
    Molecular Biology of Neurodegenerative Diseases

    Molecular Biology of Neurodegenerative Diseases

    - Volume 107

    Forfatter: info mangler
    Bog
    • Format
    • Bog, hardback
    • Engelsk

    Beskrivelse

    Neurodegenerative diseases result in progressive degeneration and / or death of nerve cells which leads to problems with movement and mental functioning. Examples include Parkinson’s, Alzheimer’s and Huntington’s disease. Much research is taking place to try to identify ways to prevent or lessen the impact of these diseases. This volume reviews the latest research and developments in the molecular biology of neurodegenerative diseases.

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    Detaljer
    • SprogEngelsk
    • Sidetal498
    • Udgivelsesdato22-05-2012
    • ISBN139780123858832
    • Forlag Academic Press Inc
    • FormatHardback
    Størrelse og vægt
  • Vægt730 g
    • coffee cup img
    10 cm
    book img
    15,1 cm
    22,9 cm

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    Als Scanning Calcium Dementia Meta-analysis Cell death Yeast Oligomers Saccharomyces cerevisiae Polymerization Phosphorylation Amyloid Cerebrovascular Disorders Mitochondria Apoptosis Protein folding Autophagy Molecular chaperones Aggregation Huntington Disease Cerebral Hemorrhage Alzheimers Neuroimaging PET Membranes Parkinsons Genome-Wide Association Study Alzheimer's disease Lipid bilayer Posttranslational modification Parkin Neuropathology Molecular Chaperone Proteasome Stress response Prion PINK1 Neurotoxicity Protofibrils Mouse models Neurodegeneration Tauopathies Protein homeostasis Therapeutic approaches MRI Mitochondrial Dysfunction Unfolded Protein response VEGF Protein misfolding Misfolding Tau Excitotoxicity Polyglutamine Pores Channels Adenine nucleotide translocator Amyloid β-protein Brain amyloid amyloid fibril Amyloid ligands Cerebral amyloid angiopathy Causal genes Fibrils Cyclophilin D DJ-1 EAAT2 microtubule Mitochondrial permeability transition pore Motor neuron β-Sheet Glutathionylation Huntingtin [PSI+] prion proteolytic cleavage α-synuclein [URE3] prion Voltage-dependent anion channel [PIN+] prion ppif SH3 Domain transcriptional dysregulation Perivascular drainage pathway Risk genes SOD1

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